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Glutathione prevents high glucose-induced pancreatic fibrosis by suppressing pancreatic stellate cell activation via the ROS/TGFβ/SMAD pathway | Cell Death & Disease
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You can never be absolute in biology—it’s not mathematics But Peter would say the answer to that question is no The chance of what you described is about as close to zero as it gets A drug can have broad effects, but only when it hits a fundamental pathway and it earns that breadth For example, let’s look at GLP-1 agonists They didn’t arrive trying to fix diabetes, obesity, heart disease, kidney disease, fatty liver, sleep apnea, and neurodegenerative disease all in one shot They got 1 indication, proved it in rigorous trials, and then over years as real evidence came in Then they earned additional ones BPC-157 has done the exact opposite They purported benefits and have expanded, while the first claim was never nailed down Wound healing became tendons, then ligaments, then muscle, then gut disease Then inflammation, pain, performance, recovery, even multiple sclerosis That’s not a new drug earning indications “ That’s a peptide getting repositioned across every therapeutic area imaginable, with nothing rigorous to show for a single human disorder
