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The AKT/mTOR hyperactivation in these organoids suggests that mTOR inhibitors (e.g., rapamycin analogs) may ameliorate synaptic imbalances, warranting clinical exploration in ASD subtypes with CNTNAP2 mutations

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In this context, BPC 157 was found to antagonize the anxiety behavior triggered by amphetamine, whose activity and effects were due to elevated extracellular dopamine levels [55,101]